The World Health Organization has called obesity a global epidemic . Indeed, its prevalence is increasing worldwide at alarming rates in adults and children . This marked and rapid increase in the prevalence of obesity is found in affluent countries, in countries in economic transition and in developing countries. This increase in prevalence in all settings suggests the important contributions of both exogenous factors in addition to genetic predisposition . The identification of contributing factors is important because obesity in children has marked adverse effects in childhood, adolescence and adulthood. Its ad-verse long-terra effects extend well into adulthood. Obese children tend to suffer from psychosocial distress and, in many environments, from considerable discrimination. Long-term prospective studies of obese adolescent females indicate that they achieve fewer years of education, and in adulthood have higher rates of poverty, lower rates of marriage and less household income than do those who were normal weight in adolescence [4, 5]. Obesity negatively influences cardiovascular risk factors such as dyslipidaemia, glucose intolerance and arterial hypertension. Dyslipidemia characterized by increased concentrations of plasma triglycerides and low density lipoprotein (LDL) cholesterol, and reduced high density lipoprotein (HDL) cholesterol concentrations, is a common finding in young obese children [6, 7]. Obesity induces reduced insulin sensitivity, pathological glucose tolerance and increased fasting as well as post-prandial blood glucose concentrations.